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This synergistic interaction enhances resilience against oxidative stress and supports continuous redox cycling in experimental models of aging and cellular defense
10.1016/j.ejmech.2017.11.057 32 KaravolosS.ReynoldsM.PanagiotopoulouN.McElenyK.ScallyM.QuintonR
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Enhanced expression of egr-1 was combined with a multitude of pathologies (i.e., atherosclerosis [159], stenosed calcific valvular disease [161], cardiac hypertrophy [162,163], cerebral ischemia [164], and tumor progression [158,165,166,167])
