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Guillain-Barr syndrome after rituximab in a patient with idiopathic thombocytopenic purpura: a causal association
Emerging evidence suggests that inhibition of the Nrf2/Keap1/ARE pathway provides chemotherapeutic effects against cancers including brain cancer [146, 154, 155]
Typically, FRs are attached to the plasma membrane of cells via GPI-anchor and can be released from the membrane by a specific enzyme, the so-called phospholipase C
The extracellular polymeric compounds that encase these cells, often consisting of proteins, eDNA, and polysaccharides, exhibit strong antibiotic resistance
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