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CD5/ LNP-FAPCAR 2.2, IgG/ LNP-FAPCAR 2.2, or saline was injected into BLM mice at postoperative days 7 and 14

This observation further supports the pivotal role of the CINC-3/CXCR2 signaling axis in sustaining persistent neuroinflammation following traumatic peripheral neuropathy

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