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Ann Vasc Surg 20(4):435-9, 2006

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Conclusion Molecular mechanisms underlying the resistance of pituitary tumor to pharmacological treatment with DA and SS analogs might involve different steps that from the receptor activation by agonist lead to the final biological responses in terms of hormone secretion inhibition as well as tumor shrinkage

demonstrated that Erastin treatment in fibroblasts and fibrosarcoma cells robustly induced the conversion of LC3I to LC3II and the formation of GFP-LC3 puncta, which are definitive hallmarks of autophagy activation (49)