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In addition, T2D is the result of a complex interaction of environmental and genetic factors, being difficult to establish the role of each one in the pathogenesis of diabetes and in the levels of IGF-1

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Brief treatment with a low dose of romidepsin on Shank-3 mice led to the prolonged restoration of social behaviors for three weeks (equivalent to several years in humans) [94]

Regulatory authorities advise assessing how renal and hepatic impairment affects the pharmacokinetics of new drugs, as this evaluation may indicate the need for dose adjustment in these populations [12,13,14,15]

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