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Both stimulate growth hormone release, but they operate through different receptor mechanisms and have distinct pharmacokinetic profiles

In this study, we showed that Tim-AIII suppressed cell proliferation and migration, induced G2/M phase arrest and ultimately triggered cell death of non-small cell lung cancer (NSCLC) cell lines accompanied by the release of reactive oxygen species (ROS) and iron accumulation, malondialdehyde (MDA) production, and glutathione (GSH) depletion

On the other hand, some treatments, like RevA, BB0689, rOspA , OspA-Ferritin, lipidated rOspA, CspZ-VLP, and OspA mRNA-LMP, did not show any significant effects

The highest amount of iron under no-stress conditions was observed with the treatment of 100 mg/L CeO 2 NPs and the lowest was observed at the highest level of stress without CeO 2 NPs, respectively (Table 7)