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Its expression is transiently turned down when new vessel formation is required, for example during wound-healing
The authors thank the members of the Data Safety and Monitoring Board, Boston, MA, and Bangalore, India (Ellis Neufeld, Chair, Leslie Kalish, Arun Mhaskar, and Kishore Phadke)
This means that the inactive form of HGF or HGF fragments can interact with Met by binding to the high affinity site but they cannot induce Met signaling because they are unable to promote receptor dimerization
But its an interesting one, Alder says
M., & Hassan, H
