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Treatment of the condition using exogenous miR-34c and miR-1 has been shown to suppress MET expression by preventing its binding to 3-UTR and thereby inhibiting prostate cancer's aggressive phenotype (Hagman et al., 2013

A frameshift mutation in ARMC3 is associated with a tail stump sperm defect in Swedish Red (Bos taurus) cattle

doi: 10.1186/s12964-022-00909-0

This significantly increased the C max and AUC of ginsenosides Rb1, Re, Rg1, ferulic acid, astragaloside IV, and paeoniflorin

2005;289(5):F1040F1047
