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Furthermore, MHC class II-dependent microglia antigen presentation seems dispensable for disease establishment and progression, as observed in EAE and cuprizone models (145), corroborating to a less prominent role of these cells, at least, in triggering inflammation
doi: 10.1161/JAHA.120.020358
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In male rat POS-offspring, supplementation with capsaicin (P56-70) [148], VA(P46-60) [149], MFGM (P21-W13/14) [90] or quinoa supplemented food (P21-P52/53) [152] improved ES-induced cognitive impairment at P63-70 in the ORT and BM [148] and in the SBT at P60 [149], the MWM, but not the ORT at W7-13 [90] and in the YM at P52-53 [152]
This proceeds via the enzyme methionine synthase reductase (MTRR), which catalyzes the reaction of 5 methyl tetrahydrofolate (5 MeTHF) to THF, and transfers one methyl group to B12
