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10.2147/DDDT.S82030 [DOI] [PMC free article] [PubMed] [Google Scholar] Ilic, S
concurrently, BNIP3 recruits Parkin to mitochondria to initiate autophagic flux, whereas NIXas a ubiquitination substrate of Parkinundergoes conformational remodeling post-modification to specifically engage adaptor protein NBR1, ultimately enabling targeted autophagic degradation of compromised mitochondria (Wang et al., 2023)
This approach aims for more consistent serum levels
The changes were gradual but real
