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[PubMed] [Google Scholar] 52.Madayag A, Lobner D, Kau KS, et al
In the case of oxidative stress, the redox-sensitive Keap1 is inactivated which allows the translocation of NrF2 into the nucleus to fulfill its role [57, 58]
Mitochondria, the tiny engines inside our cells, use oxygen and the absorbed nutrients to produce energy this energy is what makes our cells and tissues work, and whats behind our every muscle movement, from blinking to running
In summary, this dual role highlights that physiological H 2 S promotes Treg differentiation via epigenetic (Tet1/Tet2) and metabolic (LKB1-AMPK) pathways to maintain immune homeostasis, whereas excessive tumor-derived H 2 S over-activates Tregs through ENO1 persulfidation, driving pathological immunosuppression ( Figure 6B )
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