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Relative ratios of the spot densities between BPC-157-treated and control samples were calculated

These results suggest that the putatively vesicular copper released from neurons following NMDAR stimulation (Schlief et al., 2005) may act on NMDARs themselves to rein in excessive excitation, and that additional copper release can protect neurons from excitotoxic cell death

I also review official statements from agencies to highlight key safety and efficacy points

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