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R., Tran, K., Reddy, S
Mitochondrial dysfunction associated with increased oxidative stress and -synuclein accumulation in PARK2 iPSC-derived neurons and postmortem brain tissue
However, this phenotype indicates that unregulated GSH synthesis or high levels of GSH may disrupt certain critical aspects of development
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On the other hand, secondary metabolites are synthesized in different cellular compartments and function as signaling molecules and defense against environmental stresses (Godoy et al., 2021)
