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RAGE-mediated neutrophil dysfunction is evoked by advanced glycation end products (AGEs) J Leukoc Biol
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Serum antiphospholipid antibody levels as biomarkers for diagnosis of pulmonary tuberculosis patients
The neuroprotective mechanisms may involve modulation of inflammatory responses within the central nervous system, promotion of neuronal survival, and enhancement of axonal regeneration and synaptic plasticity

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