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doi:10.1016/j.redox.2015.07.012 Ansari M, Alikhani Y, Banafshe HR, Mohammadi E
Pramotton, F
Treatment of primary hepatocytes with the ER stressor tunicamycin (TM) upregulated UPR-responsive mRNAs, and ET cotreatment suppressed this effect (Figure 1A)
In the case of secondary hemochromatosis, we obtained explanted tissue samples of the heart with iron overload cardiomyopathy (IOC)
Schoellnast et al
