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BPC-157, by contrast, modulates growth factor pathways (VEGF, FGF, NO system) involved in tissue repair without directly activating hormone receptors (Sikiric et al., J Physiol Pharmacol 2018)

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In the ubiquitin-independent pathway, specific receptor proteins such as BCL2 interacting protein 3 (BNIP3)/ BCL2 interacting protein 3 like (BNIP3L), FUNDC1, and AMBRA1 recruit LC3 to facilitate mitochondria degradation [7]

10.1111/scd.12685 15 de BenoistB

Finally, most current studies are based on cell lines (such as ELT3) or animal models, whose tumor microenvironments and drug metabolism differ from those in the human uterus