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From a bioenergetic perspective, these exertional symptoms could be explained by the rapid depletion and slow regeneration of ATP, slow oxidative metabolism of fuel substrates, and increased oxidative stress in skeletal muscle, all secondary to impaired mitochondrial function
The compounded-pharmacy path, if it ever arrives, is a 2027 story at the earliest
Kaas) 397439 (Academic Press, 2020)
It enforces maladaptive metabolic reprogramming, corrupts mitochondrial signaling metabolites to promote exhaustion and inflammation, and disrupts dynamic balance and quality control systems, leading to the accumulation of damaged organelles
De Santi, C
