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GSH augmentation deserves investigation as a beneficial therapeutic approach for not only PD but also numerous other CNS disorders for which GSH deficiency and GSH-related enzyme deficits have been documented (multiple sclerosis, 25,26 autism, 2729 Alzheimers disease, 30,31 schizophrenia, 32,33 and bipolar disease 34 )

With the comprehensive application of ferroptosis molecular probes, gene editing tools, and artificial intelligence prediction models, treatment strategies will gradually shift from generalized inhibition to spatiotemporal precise intervention. Statements Author contributions CX: Writing original draft, Conceptualization, Methodology, Writing review & editing

Wang J, Wynshaw-Boris A