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In cardiomyocytes exposed to hypoxia, silencing of G6PD prevented antiapoptotic signaling in response to benfotiamine, indicating that the beneficial effects of benfotiamine on cardiomyocytes may be due to increased activation of G6PD
Although CCK signaling has been implicated in stress-induced anxiety, its role in anxiety vulnerability in ASD remains unexplored
PLoS One 8:e55407
