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Discussion Our findings indicate that combined late-life dysregulation of glutathione and iron may trigger ferroptosis in C

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During fetal and early postnatal life, adequate maternal thyroid hormone supply is critical for neurogenesis, neuronal migration, synaptogenesis, and myelination ( The entry of thyroid hormones into the central nervous system depends on specialized transporters at the BBB ( Within the brain, deiodinase enzymes modulate local hormone availability (56)

176,177 When GLP-1 binds to GLP-1R, it triggers the activation of G-proteins, leading to an increase in the intracellular second messenger cAMP
Williams LD et al
