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Since COPD is a complex and multifaceted disease, the concepts of phenotypes (i.e., subgroups with prognostic impact) and endotypes (i.e., conditions defined by specific pathophysiological and molecular mechanisms) have begun to be applied to achieve the goal of personalized medicine and a more efficient diagnostic and therapeutic approach

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However, we have previously observed similar effects with CPT1 and CPT1A inhibition considering inflammatory- and oxidative stress levels in other animal models mimicking central nervous system diseases using female rodents 28,29,30,82

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