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These toxic macromolecules enter the nucleus of neuronal cells and bind and interact with DNA, while their amplified polyglutamine tracts cause transcriptional dysregulation of antioxidant genes in the binding areas (Hensman Moss et al

Adoptive transfer The LT CD4+ cells were isolated from the spleens of BALB/c expressing a congenic Ly5.1 marker mouse after the acute respiratory allergy to HDM protocol as described previously

Dysbiosisan imbalance of commensal and pathogenic microbesis increasingly recognized as a trigger for autoimmune disease, including thyroid autoimmunity
doi: 10.1016/j.tube.2006.01.006 178 RoyEStavropoulosEBrennanJCoadeSGrigorievaEWalkerBet al

In addition, diseases related to mitochondrial dysfunction as well as therapies targeting mitochondria are also included
