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Signaling via NF-B and MAPK pathways drives pro-inflammatory cytokine gene expression (37), while a concurrent shift to aerobic glycolysis stabilizes HIF-1, which further amplifies the transcription of cytokines like IL-1 (63, 66)

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Similarly, significantly elevated NQO1 protein levels were shown in HFD-fed mice as well as in HepG2 cells treated with oleic acid as compared to respective controls (Zhang et al

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83,84 These translocated microbial components act together to transform local intestinal disturbances into systemic and sustained hepatic inflammatory attacks