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A mechanistic approach to explore novel HDAC1 inhibitor using pharmacophore modeling, 3D- QSAR analysis, molecular docking, density functional and molecular dynamics simulation study

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Triclosan (TCS) promotes microglial activation and inflammatory cytokine release by increasing PKM2 expression in BV-2 cells, further supporting PKM2s role in driving neuroinflammation through metabolic reprogramming [30]

Ginsenoside Rb1 inhibits fibrillation and toxicity of alpha-synuclein and disaggregates preformed fibrils