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Therefore, targeting the involvement of NRF2-GPX2-NOTCH3 axis holds potential as a therapeutic strategy for treating HNSCC characterized by NRF2 activation
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In the vasculature, oxidative stress is commonly associated with proteolysis, glycocalyx degradation, increased permeability, and endothelial dysfunction due to the scavenging of NO by ROS, which diminishes NO bioavailability, impairs vasodilation and promotes inflammation (Figure 8)
