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This restoration of GABAergic signaling helped in reducing glutamate excitotoxicity, nevertheless the improvements were modest compared to the effects observed with combination therapy

this angiogenesis can be inhibited by miR-29 overexpression, leading to a downregulation of B7H3 (242)

10.1186/s13287-019-1304-z 50 QuintanillaM

Oral administration of sepiapterin, a precursor to BH4, has demonstrated elevated plasma levels of BH4 in rats and alleviated BLM-induced PF [132]
