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Work from our and other groups suggested LAC as a novel epigenetic modulator of brain plasticity and a therapeutic target for clinical phenotypes of depression linked to childhood trauma

Residues Y239 (TM5), Y358 (TM7) and F443 (TM10) all positioned ideally to form strong cation-\(\pi\) interactions, with nearby residues Y211, F359, Y387 and Y447 positioned within reasonable distance to form weak cation-\(\pi\) interactions

Promotes the uptake of acetyl CoA into the mitochondria during fatty acid oxidation

As evidence accumulates supporting its use in neurodegenerative diseases and nerve regeneration, pharmaceutical companies are investing heavily in developing prescription-grade formulations