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Through integrated proteomic and metabolomic analyses, we identified a high-inflammation, high-oxidation, low-metabolism triad in the hippocampus of CLP-induced SAE mice, providing novel insights into mechanisms underlying persistent cognitive deficits in SAE survivors

Antioxidants in critical illness

however, no phylogenetic or functional analysis of the proteins, including detailed genetic background, has yet been available

doi: 10.1111/jpi.12828

These effector T cells and their cytokines have then been implicated in neuronal injury and glial activation in diseases such as AD and PD through direct cytotoxic effects or through recruitment of immune cells and inflammation (Shi et al., 2022)