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Ma D, Gilbert T, Pignanelli C, Tarade D, Noel M, Mansour F, et al
The approaches are designed to recognize and bind to specific mtDNA sequences, induce a double-strand break, and initiate targeted degradation of the mutant mtDNA in a heteroplasmic population
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In addition, accumulation of apoC-III-rich apoB-containing lipoproteins might have direct atherogenic consequences
