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HARRILL, LINDA J., Spindale, N

Lohrasbi, S., Mirzaei, E., Karimizade, A., Takallu, S
Meanwhile, ATP signaling via the same receptor enhanced the release of profibrotic tissue-metalloprotease inhibitor 1 (TIMP1), which attenuates the function of ECM-degrading enzymes, resulting in increased interstitial amounts of ECM, a hallmark of lung fibrosis ( 2+ and ATP are well-known stimulating agents of surfactant secretion (Voyno-Yasenetskaya et al., 1991

Loriot, Y

Xia R, et al