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110 Despite a number of studies that reported somewhat conflicting results regarding the different cardiac phenotypes in response to increased AKT activity, it has currently been recognized that short-term appropriate AKT activation leads to physiological cardiac hypertrophy, while long-term AKT activation or AKT overexpression causes pathological cardiac hypertrophy, interstitial fibrosis, and even heart failure

(2010) demonstrated that NF1 knockdown increased ERK signaling and increased OPC proliferation in the developing spinal cord (109)

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PMID: 35150150