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Its pharmacological effects stem primarily from the disruption of monoaminergic neurotransmission and its downstream neurotoxic consequences

Hooper and Mr

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elegans and mammalian organisms that are critical for protection from hypoxia (Pursiheimo et al., 2009

Cancer cachexia induces major metabolic disruptions, including alterations in lipid metabolism and glycometabolism, that contribute to the aggravation of cachexia symptoms [45]