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Nanomaterials-induced programmed cell death: focus on mitochondria

For acute OIPN, the dysfunction of ion channels, OCT protein, and glial cells are involved in the acute pain, whereas the main mechanisms relevant to chronic OIPN are nuclear DNA damage, oxidative stress-induced mitochondrial damage, glia activation-related neuroinflammation, and gut microbiota-induced inflammation (Fig

In contrast, human IPF is a chronic and progressive disease that may take months or even years to develop and progress to irreversible fibrosis [6]

The NLRP3 inflammasome also interacts with NF-B and induces transcription of pro-inflammatory cytokines which promotes further activation of NLRP3 (42)

Some studies demonstrated that reduced level of melatonin in AD brain contributes to the cognitive decline in AD patients and also is linked to the pineal gland volume [35, 37, 38]