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This cellular priming provides the mechanistic foundation for FOXO4-DRI's selective targeting, enabling researchers to eliminate senescent cells without affecting healthy tissue function
Genetically determined differences in the expression of these enzymes could explain interindividual risks in developing acquired aplastic anemia
The biological effects of TH are mediated through its binding to thyroid hormone receptors (TRs), which initiate downstream genomic and non-genomic pathways
