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Science 248: 596 Orlowski M, Meister A

8,9 The increasing number, mechanisms of action, variable efficacy, and overall complexity of therapies means that any physician managing UC requires a nuanced understanding of therapeutic application

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55 , 56 Mutations or deficiencies in sphingolipid degradation pathways can result in the accumulation of sphingolipids, thereby compromising lysosomal stability and resulting in an array of pathologies, including a pro-inflammatory phenotype that promotes neurodegeneration

The authors showed that SlEIL2 repressed the expression of the gene L-GALACTOSE-1-PHOSPHATASE 3 ( SlGPP3 ) and MYO-INOSITOL OXYGENASE 1 ( SlMIOX1 ) at the transcriptional level, resulting in a 1.6-fold increase in Asc synthesis through the L-galactose and myo-inositol pathways in the mutants (Table 2) [67]