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This series of pathological alterations further exacerbates local hypoxia and, through the classic hypoxia signaling pathway, stabilizes HIF-1, thereby driving disease progression [102]

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Therefore, determining the appropriate dosage to alleviate TAO-related orbital lesions without inducing adverse effects is an important area for future research

Increased hemichannel activity is associated with the release of glutamate and ATP, which can trigger excitotoxicity 479

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