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Moreover, these variants suppressed phosphorylation of SHP2 and ERK to a significantly greater extent than STAT3, with the D23L/S117N variant in particular reducing both SHP2 and ERK signaling by approximately 90%, despite retaining approximately 50% STAT3 activation relative to treatment with WT leptin alone (Fig
Hill DB, Vasquez PA, Mellnik J, McKinley SA, Vose A, Mu F, et al
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