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Description
Functional impairment in Miro degradation and mitophagy is a shared feature in familial and sporadic Parkinsons disease
The isolated E-peptides did modulate ERK1/2 and Akt phosphorylation and increased satellite-cell proliferation, and the Eb-type peptide drove significant muscle hypertrophy but that hypertrophy was IGF-1 receptor-dependent, and, critically, the added muscle mass came at the expense of muscle strength
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The expression, induction and pharmacological activity of CYP1A2 are post-transcriptionally regulated by microRNA hsa-miR-132-5p
