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Clinical data from LS patients also highlight marked gliosis as a characteristic finding, potentially resulting from reactive processes secondary to neuronal damage but it could be also a result of an increased propensity of NPCs to differentiate into astrocytes due to LS-causative mutations and mitochondrial dysregulation

Journal of the International Society of Sports Nutrition, 11 (1), 4

This is especially so, as we observe that dimeric CBS 516525 also fits the two-site model even though this construct cannot form higher oligomers (Supplementary Figs

[DOI] [PMC free article] [PubMed] [Google Scholar] 29.Li X, Chou YT, Husain R, Watson JT

Targeting Mitochondrial ROS-Mediated Ferroptosis by Quercetin Alleviates High-Fat Diet-Induced Hepatic Lipotoxicity