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Activation of signaling molecules downstream of TLR4, including NF-kB and those mediated by MAPK kinases such as JNK, ERK and p38, results in the transcription of numerous pro-inflammatory genes, including TNF-, IL-6, IL-1, and COX-2, subsequently eliciting a UC inflammatory response (Wang G

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[DOI] [PMC free article] [PubMed] [Google Scholar] 106.Edmans J.G., Murdoch C., Hatton P.V., Madsen L.S., Santocildes-Romero M.E., Spain S.G., Colley H.E