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Triiodothyronine-induced accumulations of malic enzyme, fatty acid synthase, acetyl-coenzyme A carboxylase, and their mRNAs are blocked by protein kinase inhibitors
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327,328 This disruption contributes to nerve injury by affecting mitochondrial function and axonal trafficking
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[37] [38] [202] Notably, amphetamine and trace amines possess high binding affinities for TAAR1, but not for monoamine autoreceptors
