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The administration of liproxstatin-1 or ferrostatin-1, which are specific compounds that suppress ferroptosis, shields intestinal epithelial cells from peroxidative damage and aids in the recovery of intestinal barrier integrity (173, 174)

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Below, we will focus on the critical role of GSH, without discarding that OxS could also result from dysregulation of other antioxidant systems (thioredoxin [19], peroxiredoxin, sulforedoxin [20])

BPC-157 appears to influence multiple aspects of this healing cascade