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The analogues, 1,8-diMQ and 5-amino-1MQ were tested at concentrations from 200 M 10 nM and 600 M 10 nM, respectively

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Consistently, GGCT knockdown reduced mitochondrial respiratory capacity (basal, maxillary and reserve respiration) in C4-2 cells compared with the control group (Fig

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Disruption of the tight endothelial junction can be achieved via the use of a hyperosmotic agent to produce osmotic disruption or use of a vasoactive compound, bradykinin analogs, or other molecules [3]