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Collectively, these findings implicate sustained dysregulation of CCK within the mPFC as a mechanistic substrate of stress-induced anxiety susceptibility in ASD

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A systematic review found no acute toxicity in animals and no adverse events across various doses

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Experimental literature has also investigated VEGF and VEGFR2 signaling, growth hormone receptor expression in tendon fibroblasts, neurotransmitter modulation, and neuroprotective pathways involving the dopaminergic and serotonergic systems