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It is notable that high AR activity, rather than high AR expression per se, has been identified as a predictive biomarker of response to this treatment and transcriptional downregulation of AR has emerged as a mechanism of resistance to HD androgens in a subset of patients 33

Astrocytes are also involved in the recruitment of inflammatory cells to the site of injury, through the formation of a glial scar termed reactive astrogliosis which involves activation and proliferation of astrocytes to limit extending damage ( Bmal1 , shown through activation and increased inflammatory gene expression using an astrocyte specific Bmal1 knockout ( Bmal1 knockouts in astrocyte cultures have reported reduced actin-binding protein cortactin and impaired actin stress fiber formation ( Other immune cell populations that are involved in regulating the progression of ischemic injury are neutrophils, macrophages, and T-cells ( Bmal1 expression (Scheiermann et al., 2013)