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We measured the response to ADP stimulation in young and old muscle mitochondria with ELAM treatment, in vivo heart and muscle function, and compared protein abundance, phosphorylation, and S-glutathionylation of ADP/ATP pathway proteins

Usually, damaged melanocytes are eliminated through apoptosis and senescence, but research by Ken et al suggests that MSCs depletion may occur due to ectopic differentiation and impaired renewal

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