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For instance, inhibition of apolipoprotein C1 (APOC1) or SLC7A11 promotes ferroptosis in TAMs, which is characterized by increased iron content, downregulated anti-ferroptosis mediators (GPX4, NRF2, SLC7A11, and GSH), and significant ferroptosis-associated mitochondrial changes

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PABA/NO as an anticancer lead: analogue synthesis, structure revision, solution chemistry, reactivity toward glutathione, and in vitro activity

LPCAT3, a key player in the ferroptosis mechanism of cells, facilitates the incorporation of PUFAs into phospholipids, which are essential substrates for lipid peroxidation in ferroptosis (129)