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Experimental evidence suggests that PQQ promotes lipolysis inside mitochondrial matrix through -oxidation to release free fatty acids could be a potential source of energy evidenced in an in vivo model (Jonscher and Rucker, 2019)
Consequently, it resists the onset of ferroptosis
doi: 10.1016/j.bj.2023.100677 144
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These findings further ruled out mitochondrial damage to be the source of GSH depletion-induced oxidative stress