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Description
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Most published studies assume that, like canonical CPT1 isoforms, CPT1C is a mediator of fatty acid transport to mitochondria for beta-oxidation, despite the fact that CPT1C has inefficient catalytic activity and is located in the endoplasmic reticulum

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Researchers have explored similar multi-peptide combinations in experimental models relating to cellular regulation, peptide-mediated signalling behaviour, and broader biochemical processes under controlled laboratory conditions
