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In both aging and the response to hydrocarbons, the common pathological factor may be the accumulation of alpha2u-globulin, via a susceptible pathway not shared with other proteins (Neuhaus, 1986

Deficiency in vitamin B12 can lead to the accumulation of methylmalonyl-CoA, which is subsequently converted to methylmalonic acid (MMA) (, , )

This condition was first described in 1966 (76) and may present as a subacute or acute encephalopathy with seizures and stroke-like episodes, often in association with myoclonus and tremor (77)

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